Dominant Eye Test

The dominant eye is the eye that looks directly at an object. The non-dominant eye is the eye that looks at the same object at a slight angle. This small difference provides depth perception.

Being right or left handed will not necessarily determine if you are right or left eye dominant.

Eye dominance is an important consideration for monovision correction to reduce the need for reading glasses or bifocals. Anyone who is presbyopic should consider monovision.

Determining the dominant eye is actually very simple. We have developed a free USAEyes Dominant Eye Test Card that you can download and print from your computer.

After printing the card, cut away the dotted lines. Hold the card at arm's length and focus on a stationary object through the center hole. While staying focused on the object, bring the card close to your face. The card will automatically center on the eye that is dominant. Feel free to distribute the USAEyes Dominant Eye Test Card to your friends.

Another similar test for eye dominance is with the both eyes open, raise your right arm and point to an object in the distance. Anything more than about 20 feet away is ideal. Continue to look at and point at the object and cover the left eye. Did your pointing finger seem to move off the target? Continue pointing and cover your right eye. Did your pointing finger seem to move off the target with your right eye covered? Your dominant eye is the eye you using when the pointing finger does not seem to move. Being right or left handed will not necessarily determine if you are right or left eye dominant.

Try both tests. They should both determine the same eye is dominant.

If you are ready to choose a doctor to be evaluated for conventional or custom wavefront Lasik, All-Laser Lasik, PRK, LASEK, Epi-Lasik, NearVision CK, RLE, or any refractive surgery procedure, we highly recommend you consider a doctor who has been evaluated and certified by the USAEyes nonprofit organization. Locate a USAEyes Evaluated & Certified Lasik Laser Eye Surgeon.

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Current Dominant Eye Medical Journal News...

Genetic evidence for a role for protein kinase A in the maintenance of sleep and thalamocortical oscillations.

Related Articles

Genetic evidence for a role for protein kinase A in the maintenance of sleep and thalamocortical oscillations.

Sleep. 2010 Jan 1;33(1):19-28

Authors: Hellman K, Hernandez P, Park A, Abel T

STUDY OBJECTIVES: Genetic manipulation of cAMP-dependent protein kinase A (PKA) in Drosophila has implicated an important role for PKA in sleeplwake state regulation. Here, we characterize the role of this signaling pathway in the regulation of sleep using electroencephalographic (EEG) and electromyographic (EMG) recordings in R(AB) transgenic mice that express a dominant negative form of the regulatory subunit of PKA in neurons within cortex and hippocampus. Previous studies have revealed that these mutant mice have reduced PKA activity that results in the impairment of hippocampus-dependent long-term memory and long-lasting forms of hippocampal synaptic plasticity. DESIGN: PKA assays, in situ hybridization, immunoblots, and sleep studies were performed in R(AB) transgenic mice and wild-type control mice. MEASUREMENTS AND RESULTS: We have found that R(AB) transgenic mice have reduced PKA activity within cortex and reduced Ser845 phosphorylation of the glutamate receptor subunit GluR1. R(AB) transgenic mice exhibit non-rapid eye movement (NREM) sleep fragmentation and increased amounts of rapid eye movement (REM) sleep relative to wild-type mice. Further, R(AB) transgenic mice have more delta power but less sigma power during NREM sleep relative to wild-type mice. After sleep deprivation, the amounts of NREM and REM sleep were comparable between wild-type and R(AB) transgenic mice. However, the homeostatic rebound of sigma power in R(AB) transgenic mice was reduced. CONCLUSIONS: Alterations in cortical synaptic receptors, impairments in sleep continuity, and alterations in sleep oscillations in R(AB) mice imply that PKA is involved not only in synaptic plasticity and memory storage but also in the regulation of sleep/wake states.

PMID: 20120617 [PubMed - in process]